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By T. H. Crook III Ph.D., G. J. Larrabee (auth.), Prof. Dr. Lüder Deecke, Prof. Dr. Peter Dal-Bianco (eds.)

The papers compiled during this supplementum are a variety of the easiest con­ th tributions provided on the 19 Central-European Neurological Symposium (CNS 19) hung on June 29 - July 1, 1989 in Vienna. the most subject of this convention was once degenerative and age-associated neurological ailments. In fresh many years lifestyles expectancy has dramatically elevated, a minimum of within the industrialized international locations. This has ended in severe distortions of the so-called inhabitants pyramids that now not seem like such yet start to nearly resemble cylinders. as a result of this "overaging" of the inhabitants, illnesses which are linked to age became even more universal than prior to. It was once therefore greater than moderate to dedicate a congress of the CNS sequence to those vital neurological ailments. the subsequent fields of curiosity are coated: Age-associated reminiscence impairment (AAMI), Alzheimer's and different dementias, Parkinson's illness and different circulation problems, stroke and others. in regards to the de males­ tias, a few papers care for prognosis utilising neuro-imaging tools resembling MRI, CT, puppy and SPECT, others utilizing electrophysiological equipment. a major point within the early preclinical prognosis of dementia is the inclusion of neuropsychological checks to reinforce the opportunity of effec­ tive early therapy. additionally medicines which are now lower than medical research are mentioned and initial effects are presented.

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1. Morphometric evaluation of the human corpus callosum (for details and abbreviations see text) the horizontal inferior 1 and tangent to the most superior point of the trunk (Fig. 1). For further methodological details see Weis et al. (1989). The result of this procedure was a rectangular figure which enabled us to measure a) callosal height (CH) as the distance between HI -1 and HS-1 and b) callosal length (CL) as the distance between VA-1 and VP-1 (Fig. 1). The callosal length was divided into two, three, four and five parts by lines which were traced perpendicular to the HI -1 and HS-1 (Fig.

Ubiquitin reactivity was generally absent in early stage I-tangles. , 1987). By silver stain these neurones did not exhibit NFT. Ultrastructurally tau reactivity in such neurons was associated to single PHF. These data thus suggest, that immunostaining with tau antibodies renders the recognition of a very early stage of tangle formation, which precedes the morphological appearence of argyrophilic fibrillar inclusions. This pattern of NFT immune reactivity we named "stage 0" tangles. These data could either mean that with maturation of tangles more and more ubiquitin is incorporated into the fibrils (ubiquitination), which finally 30 F.

Rev Neurol 145: 443-450 Li G, Shen YC, Che CH, Zhao YW, Li SR, Lu M (1989) An epidemiological survey of age-related dementia in an urban area of Beijing. Acta Psychiatr Scand 79: 557-563 Liston EH, La Rue A (1983a) Clinical differentiation of primary degenerative and multiinfarct dementia: a critical review of the evidence. Part I. Clinical studies. Bioi Psychiatry 18: 1451-1465 Liston EH, La Rue A (1983b) Clinical differentiation of primary degenerative and multiinfarct dementia: a critical review of the evidence.

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