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By M. V. Singer, D. Brenner

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Additional resources for Alcohol And the Gastrointestinal Tract: Special Issue: Digestive Diseases 2005

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In rats, obstruction of the pancreatic duct was achieved with ethibloc application, a tissue adhesive which is totally decomposed by the organism within 2 months [81]. Application of ethibloc alone resulted in pancreatic injury with extensive fibrosis, inflammatory cell infiltration, and acinar cell degeneration. However, these injuries were reversible after ethibloc decomposition. Further prolonged alcohol administration with an intragastric cannulae inhibited the recovery, and resulted frequently in parenchymal calcifications.

21 Kvietys PR, Twohig B, Danzell J, Specian RD: Ethanol-induced injury to the rat gastric mucosa. Role of neutrophils and xanthine oxidase-derived radicals. Gastroenterology 1990; 98:909–920. 22 Szabo S, Trier JS, Brown A, Schnoor J: Early vascular injury and increased vascular permeability in gastric mucosal injury caused by ethanol in the rat. Gastroenterology 1985;88:228– 236. 23 Iwata F, Zhang XY, Leung FW: Aggravation of gastric mucosal lesions in rat stomach by tobacco cigarette smoke. Dig Dis Sci 1995;40: 1118–1124.

Other studies confirmed that there was a decreased risk of oral, pharyngeal and esophageal cancers among California Seventh-Day Adventists, based on mortality data. Cancer incidence and mortality among Mormons have also been studied. In Utah, where detailed cancer incidence rates are available, the incidence of cancer of the oral cavity, pharynx, esophagus and larynx was 20–40% among Mormons compared with non-Mormons [36]. In California, mortality rates among Mormon men were 53% for oral-pharyngeal, 45% for esophageal and 30% for laryngeal cancer, compared with the general population of the USA [37].

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